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Amixide (Amitriptyline)

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Amixide is not FDA-approved to treat insomnia, but one of the side effects of the medication is sleepiness, especially when you first start taking it. Because of this, you may want to take it in the evening or before bedtime when you first start taking it.

Other names for this medication:
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Also known as: Amitriptyline


Amixide (amitriptyline) is a tricyclic antidepressant with sedative effects. Amitriptyline affects certain chemical messengers (neurotransmitters) that communicate between brain cells and help regulate mood.

Amixide is a prescription medicine used to treat symptoms of depression.


Usual Adult Dose for Depression:

OUTPATIENTS: -Initial dose: 75 mg orally per day in divided doses; this may be increased to 150 mg/day (if needed) -Maintenance dose: 40 to 100 mg orally per day -Maximum dose: 150 mg/day Alternate outpatient treatment regimen: 50 to 100 mg orally as a single dose at bedtime; this may be increased by 25 or 50 mg as needed at bedtime to a total of 150 mg/day INPATIENTS: -Initial dose: 100 mg orally per day -Maintenance dose: 40 to 100 mg orally as a single dose at bedtime -Maximum dose: 300 mg/day Comments: -Dose increases should preferably be made in the late afternoon or at bedtime due to the sedative effect. -The full therapeutic effect may take as long as 30 days to develop. -Maintenance doses should be reduced to the lowest amount that will maintain relief of symptoms when satisfactory improvement has been obtained. -Maintenance therapy should be continued for 3 months or longer to lessen the possibility of relapse. Use: Relief of symptoms of depression

Usual Geriatric Dose for Depression:

10 mg orally 3 times a day AND 20 mg orally once a day at bedtime Comments: -The full therapeutic effect may take as long as 30 days to develop. -Elderly patients should be monitored carefully and serum levels obtained as clinically appropriate. -Dose adjustments should be made according to clinical response. Use: Relief of symptoms of depression

Usual Pediatric Dose for Depression:

12 years or older: 10 mg orally 3 times a day AND 20 mg orally once a day at bedtime Comments: -The full therapeutic effect may take as long as 30 days to develop. -Dose adjustments should be made according to clinical response. Use: Relief of symptoms of depression

Detailed Amixide dosage information


Seek emergency medical attention or call the Poison Help line at 1-800-222-1222. An overdose of amitriptyline can be fatal.

Overdose symptoms may include irregular heart rhythm, feeling like you might pass out, seizures, or coma.


Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Amixide are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Do not take Amixide if you are allergic to Amixide components. Do not take Amixide if you are pregnant, planning to become pregnant, or are breast-feeding. Be careful with Amixide if you suffer from or have a history of a history of heart attack, stroke, bipolar disorder (manic-depression), schizophrenia or other mental illness, diabetes, overactive thyroid, glaucoma, problems with urination, heart disease, seizures. Be careful with Amixide if you are taking guanethidine (Ismelin), disulfiram (Antabuse), heart rhythm medications such as flecainide (Tambocor), propafenone (Rhythmol), quinidine (Cardioquin, Quinidex, Quinaglute), cimetidine (Tagamet). Avoid alcohol. Be careful! Taking Amixide you can become suicidal. Be careful when you are driving or operating machinery. Be careful with Amixide if you are going to have a surgery. It can be dangerous to stop Amixide taking suddenly.

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Compared to the lean controls, obese Zucker rats showed significant increases in collagen staining, as well as expressions of collagen, fibronectin, plasminogen activator inhibitor-1, and two major proinflammatory mediators (i.e., interleukin-6 and monocyte chemoattractant protein-1). Reliability of an intravenous intermittent access port (saline lock) for obtaining blood samples for coagulation studies. Ranitidine and amoxicillin for eradication of Helicobacter pylori in patients with duodenal ulcer. Improvements on current therapies for the histiocytoses will depend on continued advances in the understanding of these enigmatic diseases. Relationships between speech production and speech perception skills in young cochlear-implant users. APP1 and other known genes (PAH1, DPP1, LPP1) are responsible for all detectable PAP activity in yeast. Interventions aimed at reducing the delay in acute myocardial infarction treatment should primarily focus on the help-seeking behavior of patients. Intracellular analysis of the effects following micropolarization of cortical neurons. Therefore, we attempted at a retrospective search for causes of the failure. Interestingly, IL-24 attenuated the H(2)O(2)-induced ROS production by reducing the mitochondrial H(2)O(2) production and enhancing the expression of antioxidant enzymes.

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These events were associated primarily with the easterly trade winds and the African easterly wave that extends from the subtropical eastern Atlantic to the Caribbean. Short-term inflammatory responses following intratracheal instillation of fine and ultrafine carbon black in rats. This study reports a 38-year-old woman with adult Leigh syndrome associated with partial deficiency of the pyruvate dehydrogenase complex. Pt(II) diimine complexes bearing carbazolyl-capped acetylide ligands: synthesis, tunable photophysics and nonlinear absorption. We compared plasma phospholipid FA composition between non-stroke control and ischemic stroke in Korean population, to discern the FA that distinguishes ICAS from total ischemic stroke patients. Results correlated well with those by the manual spectrophotometric method, and hemolysis no longer interfered. Mechanical valves are used in dialysis patients due to the presumed rapid degeneration of tissue valves.

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